Brain-derived neurotrophic factor in arterial baroreceptor pathways: implications for activity-dependent plasticity at baroafferent synapses.
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Stimulation of alpha(1)-adrenoceptors reduces glutamatergic synaptic input from primary afferents through GABA(A) receptors and T-type Ca(2+) channels.
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Enhanced sensitivity to group II mGlu receptor activation at corticostriatal synapses in mice lacking the familial parkinsonism-linked genes PINK1 or Parkin.
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GABAA receptor alpha5 subunits contribute to GABAA,slow synaptic inhibition in mouse hippocampus.
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Dopamine neuron responses depend exponentially on pacemaker interval.
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Gain modulation of neuronal responses by subtractive and divisive mechanisms of inhibition.
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Axonal Na+ channels ensure fast spike activation and back-propagation in cerebellar granule cells.
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Modafinil normalized hyperreflexia after spinal transection in adult rats.
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