Signaling pathways for B cell-activating factor (BAFF) and a proliferation-inducing ligand (APRIL) in human placenta.
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Manipulating B cell homeostasis: a key component in the advancement of targeted strategies.
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Homeostatic control of B lymphocyte subsets.
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The proteoglycan (heparan sulfate proteoglycan) binding domain of APRIL serves as a platform for ligand multimerization and cross-linking.
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CD4(+)CD25(+) T-cells control autoimmunity in the absence of B-cells.
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Downregulation of RUNX1 by RUNX3 requires the RUNX3 VWRPY sequence and is essential for Epstein-Barr virus-driven B-cell proliferation.
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Transmembrane activator, calcium modulator, and cyclophilin ligand interactor drives plasma cell differentiation in LPS-activated B cells.
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BAFF is increased in renal transplant patients following treatment with alemtuzumab.
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