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Anaplasma marginaleAnaplasmaAnaplasmosisAnaplasma phagocytophilumDermacentorAnaplasma centraleTicksCattle DiseasesBacterial Outer Membrane ProteinsEhrlichiosisCattleArachnid VectorsTick-Borne DiseasesRhipicephalusAntigens, BacterialAnaplasma ovisArthropod VectorsAnaplasmataceaeEhrlichiaSalivary GlandsEhrlichia ruminantiumBacterial VaccinesBabesia bovisIxodesBabesiaAntigenic VariationAntibodies, BacterialBabesiosisMolecular Sequence DataRickettsial VaccinesBacterial ProteinsTick InfestationsTheileriasisErythrocytesBacteremiaDNA, BacterialAnaplasmataceae InfectionsAmino Acid SequenceRNA, Ribosomal, 16SSequence Analysis, DNAImmunodominant EpitopesPolymerase Chain ReactionBase Sequence

Breadth of the CD4+ T cell response to Anaplasma marginale VirB9-1, VirB9-2 and VirB10 and MHC class II DR and DQ restriction elements. (73/95)

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Serological cross-reactivity between Anaplasma marginale and an Ehrlichia species in naturally and experimentally infected cattle. (74/95)

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Functional epitope core motif of the Anaplasma marginale major surface protein 1a and its incorporation onto bioelectrodes for antibody detection. (75/95)

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The twin-arginine translocation pathway in alpha-proteobacteria is functionally preserved irrespective of genomic and regulatory divergence. (76/95)

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Comparative seroprevalences of bovine trypanosomiasis and anaplasmosis in five states of Malaysia. (77/95)

A comparative seroprevalence study on bovine trypanosomiasis and anaplasmosis was conducted. Sera of adult cattle and buffaloes of different breeds from farms from five different states in Malaysia were collected and tested for the presence of Trypanosoma evansi antibodies by CATT and Anaplasma marginale antibodies by c-ELISA. Of the 116 samples, 14.7% tested positive for bovine trypanosomiasis and 77.6% for bovine anaplasmosis.  (+info)

Expression patterns of Anaplasma marginale Msp2 variants change in response to growth in cattle, and tick cells versus mammalian cells. (78/95)

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Determining the repertoire of immunodominant proteins via whole-genome amplification of intracellular pathogens. (79/95)

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Expansion of variant diversity associated with a high prevalence of pathogen strain superinfection under conditions of natural transmission. (80/95)

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