Histological and histochemical quantification of root resorption incident to the application of intrusive force to rat molars. (1/358)

This study was conducted to investigate the nature of root resorption resulting from intrusive forces applied to the rat lower molars, by means of histological and histochemical techniques with tartrate resistant acid phosphatase (TRAP). Thirty-eight 13-week-old Wistar strain male rats were used. Intrusive force was created by a fixed appliance which was adjusted to exert an initial force of 50 g for the duration of 1, 2, and 3 weeks. The degree of root resorption and distribution of TRAP positive cells were evaluated. On the root surface, the TRAP positive scores were low in the apical regions. Significant differences in the scores were found in the inter-radicular region of the roots between the experimental and control groups for the 2- and 3-week groups. More active resorption of bone occurred during the experimental period, as denoted by greater TRAP positive scores on the bone than on the root surface. Root resorption scores in the apical root region were larger in the 2- and 3-week groups than in the 1-week group. Significant differences in the root resorption scores were also found between the 1- and 3-week groups in the inter-radicular region, indicating that intrusive force application of a longer duration may lead to a higher frequency of root resorption. It is shown that, irrespective of the level of TRAP positive cells and root resorption scores, the degree of root resorption activity is higher in the apical root region than in the inter-radicular area. These results indicate that cellular cementum may be resorbed more easily because of its richer organic components and low mineralized structure.  (+info)

Rare earth magnets in orthodontics: an overview. (2/358)

Magnets have been used in dentistry for many years. They can be made to push or pull teeth. The force they deliver can be directed, and they can exert their force through mucosa and bone, as well as within the mouth. In orthodontics they are used for intrusion of teeth, tooth movement along archwires, expansion, retention, in functional appliances, and in the treatment of impacted teeth. New 'high energy' magnets are capable of producing very high forces relative to their size. Although magnets are potentially very useful there are a number of problems that severely affect their performance; the force produced between any two magnets falls dramatically with distance, significant irreversible loss in force is seen if the magnets are heated and a dramatic reduction in force is seen if the magnets are not ideally aligned to one another. In addition, magnets corrode badly in the mouth and a robust coating is required to protect them. This paper outlines the background to high energy magnets used in orthodontics, discusses the relevant physical and biological properties of them, and reviews their applications.  (+info)

Alteration in the expression level of calbindin D28k in the periodontal ligament of the rat molar during experimental tooth movement. (3/358)

The present immunohistochemical study was designed to investigate changes in the distribution and expression level of calbindin D28k in the periodontal ligament during experimental tooth movement in the rat molar to clarify the physiological role of this protein in the ligament. In normal animals, calbindin D28k-like immunoreactivity appeared sparsely in spindle-shaped cells in the alveolar half of the periodontal ligament. Electron microscopic observations showed that these immunoreactive cells were characterized by well-developed rough-surfaced endoplasmic reticulum and phagosomes--which often contained collagen fibers--suggesting that these cells could be categorized as periodontal fibroblasts. Twelve hours following the onset of the experimental tooth movement, cells positive for calbindin D28k increased in number in the periodontal ligament, especially in the alveolar half of the pressured side. Immunoelectron microscopy showed that the calbindin D28k-immunopositive cells had morphological features similar to those of fibroblasts in the normal ligament, and that these cells occasionally made contact with immunonegative macrophage-like cells. Immunopositive cells gradually decreased in number, and the distribution of the cells and intensity of the immunoreactivity returned to normal levels by 14 days following the induction of the experimental tooth movement. The present results suggest that calbindin D28k plays an important role in the homeostasis and cyto-protection of fibroblasts in the periodontal ligament at the initial phase of experimental tooth movement.  (+info)

The effects of increasing the reverse curve of Spee in a lower archwire examined using a dynamic photo-elastic gelatine model. (4/358)

This paper describes the development and testing of a dynamic in vitro photo-elastic model for evaluating the effects of orthodontic mechanics on an entire arch of teeth. A model of a mandibular arch was made and the teeth were embedded in a gelatine material with a high level of mechanical creep which permitted tooth movement in response to orthodontic forces. The excellent photo-elastic properties of this material also facilitated the analysis of the stress distribution around the roots of the teeth. The model of a mandibular arch was used to investigate the tooth movements and stress distributions produced by increasing the reverse curve of Spee in a 0.018 x 0.025-inch stainless steel archwire. The results revealed that a 1-mm reverse curve of Spee increased the arch length by 1.6 mm, but increasing the reverse curve of Spee to 5 mm did not increase arch length further. Photo-elastic analysis showed an increased stress distribution around the roots of the incisors and molars as the reverse curve of Spee was increased in the archwire.  (+info)

Neural modulation of inflammatory reactions in dental tissues incident to orthodontic tooth movement. A review of the literature. (5/358)

This article reviews the current knowledge of the biological aspects of dental tissue changes incident to orthodontic tooth movement. The inflammatory nature of these tissue changes was first recognized in the early 1970s, and since then a number of morphological and quantitative investigations have been published in support of this view. The studies dealing with vascular and cellular dental tissue changes, as well as those concerned with inflammatory mediators present at sites of orthodontic tooth movement are systematized and presented accordingly. Special emphasis is placed upon the role of the sensory nerve fibres and their neuropeptides in the control, and development of an inflammatory process, i.e. their role in tooth movement.  (+info)

Elastic activator for treatment of open bite. (6/358)

This article presents a modified activator for treatment of open bite cases. The intermaxillary acrylic of the lateral occlusal zones is replaced by elastic rubber tubes. By stimulating orthopaedic gymnastics (chewing gum effect), the elastic activator intrudes upper and lower posterior teeth. A noticeable counterclockwise rotation of the mandible was accomplished by a decrease of the gonial angle. Besides the simple fabrication of the device and uncomplicated replacement of the elastic rubber tubes, treatment can be started even in mixed dentition when affixing plates may be difficult.  (+info)

The influence of maxillary incisor inclination on arch length. (7/358)

This ex vivo study was designed to investigate Andrews' hypothesis that there is a space implication when incisors are torqued correctly. A working model was constructed to allow acrylic typodont incisors of varying known values of inclination to be substituted into the model. The arch lengths of the various 'set-ups' were measured using a reflex microscope linked to a PC. In order to quantify the space requirement of clinical relevance for adequate incisor torque, the method was repeated by substituting replicas of patients' 'natural' incisors. For both acrylic and natural incisors it was found that, as the inclination of the teeth increased, there was an increase in all arch lengths, this being greater for the natural incisors. This larger increase for the natural incisors was related not only to their increased size, but was also dependent on the morphology of the incisor. Those incisors which were parallel-sided showed the greatest increase in arch length, whereas the incisors that were relatively triangular in shape showed the smallest increase. When the inclination of an 'average' set of 21/12 is increased by 5 degrees, an increase in the arch length of approximately 1 mm may be expected.  (+info)

Skeletal and dental changes following the use of the Frankel functional regulator. (8/358)

The purpose of this study was to assess the relative contributions of skeletal and dental components in correction of Class II division 1 malocclusions when treated with Frankel's functional regulator (FR). This was a retrospective study involving analyses of pre- and post-treatment cephalograms of 63 Class II division 1 patients treated with the FR to demonstrate the relative maxillary, mandibular, incisor, and molar movements during treatment compared with normal growth within a control group of untreated 39 Class II division 1 cases drawn from the same demographic population. All cephalograms were digitized and subjected to a Pitchfork analysis, which measured individual anteroposterior skeletal and dental changes during the period of study. It was shown that the FR was effective in treating Class II division 1 cases with the studied group being corrected to a clinically acceptable overjet and overbite of 2-3 mm. The majority of the correction came from dental movements, the most significant being the retroclination of the upper incisor teeth (mean 4.1 mm, 95 per cent CI +/- 0.44) and proclination of the lowers (mean 2.2 mm 95 per cent CI +/- 0.57). As regards skeletal correction, the most significant contribution was the restraint of normal maxillary forward growth (mean -0.2 mm, 95 per cent CI +/- 0.62) with forward mandibular growth not being a significant factor.  (+info)